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Neural Processing of Emotional Words in Youth with Autism Spectrum Disorder: An Event-Related Potential Study

This ERP study compared 20 youth with autism spectrum disorder (ages 8.5–17.7 years) to 20 typically developing peers during emotional word processing tasks. Results showed intact early-stage neural responses but significantly reduced late positive component activity in autistic youth during emotional word categorization, suggesting differences in top-down evaluative processing of emotions.

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The Therapeutic Engagement Index in Autism: A Research Framework for Understanding Intervention Responsiveness

This paper introduces the Therapeutic Engagement Index (TEI), a multidimensional framework for understanding why autistic children respond differently to comparable interventions by examining observable factors affecting therapeutic accessibility and engagement. TEI is proposed as a research construct—not a validated diagnostic tool or compliance measure—to identify modifiable conditions that enhance how children can access, tolerate, and benefit from therapeutic input within specific intervention contexts.

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Emotion- and Region-Specific Gaze Differences in Parents of Children With Autism Spectrum Disorder

A study using eyetracking compared visual attention patterns in parents of children with ASD versus parents of typically developing children while viewing emotional faces. Parents of children with ASD showed selective increased attention to the mouth region specifically when viewing threat-related emotions (anger, fear), suggesting emotion- and region-specific gaze allocation differences rather than generalized attentional changes.

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Cocoa polyphenols mitigate valproic acid-induced autism-like phenotypes in zebrafish via metabolic and proteomic mechanisms: a pilot study

This pilot study examined whether cocoa extract containing polyphenols could reverse autism-like behavioral and gastrointestinal abnormalities induced by valproic acid exposure in zebrafish. Cocoa co-treatment ameliorated hyperactivity and gut dysfunction while normalizing key metabolic and protein markers of cellular stress.