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StudiuDemențeÎncredere înaltă22.06.2026

Hypertension and Dementia: Pathophysiology and Potential Utility of Antihypertensives in Reducing Disease Burden

This review examines hypertension as a modifiable risk factor for dementia, finding that mid-life hypertension (ages 45-64) correlates with increased cognitive decline and dementia risk, while late-life hypertension shows less consistent associations. Evidence on whether antihypertensive treatment effectively prevents dementia remains conflicting across studies, likely due to methodological differences between observational and randomized controlled trials.

StudiuDemențeÎncredere înaltă22.06.2026

Transgenic Amyloid Precursor Protein Mouse Models: Limitations as AD Models but Effective Predictors of Anti-Amyloid Therapies

APP transgenic mice model amyloid pathology in Alzheimer's disease and have successfully predicted the efficacy of anti-amyloid immunotherapies, including their adverse effects like amyloid-related imaging abnormalities (ARIA). While incomplete models of full AD pathology, these early transgenic models have accurately predicted therapeutic responses in human clinical trials that reduce amyloid and slow cognitive decline.

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Can Speaking Multiple Languages Help Delay Alzheimer's Disease Symptoms?

This review examines evidence that bilingualism or multilingualism may increase cognitive reserve through executive control mechanisms, potentially delaying clinical dementia symptoms by 4-5 years despite ongoing neuropathology. While multilingual use does not prevent Alzheimer's disease development, it may compensate for brain changes and reduce lifetime disease burden in older adults.

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Proteome-wide Analysis Identifies Cortical Proteins Associated with Cognitive Resilience in Aging and Dementia

This postmortem proteomics study of 604 older adults identified 47 cortical proteins in the dorsolateral prefrontal cortex that promote cognitive resilience across different cognitive domains, using mass spectrometry analysis while controlling for Alzheimer disease and related dementia pathologies. The protein NRN1 showed the strongest association, linked to multiple cognitive abilities including semantic memory, suggesting a potential role in protecting against dementia-related cognitive decline.

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Deep Brain Stimulation for Cognitive Decline in Alzheimer Disease: Systematic Review of Clinical Evidence and Target Sites

A systematic review of 14 human studies (1994–2023) examined deep brain stimulation (DBS) targeting three brain regions to slow cognitive decline in Alzheimer disease. The fornix and nucleus basalis of Meynert showed promise in slowing decline according to standardized cognitive scales, though sample sizes were modest and long-term efficacy remains to be established.